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In nerves uncovered to 100 Hz when bathed in 10 mM glucose, the CAP failed suggesting that even within the presence of normoglycemic glucose additional power substrate is required when the tissue power demand will increase. Such data are necessary as they present that glycogen has an vital function below physiological situations. The fact that glycogen supported CAP conduction within the absence of exogenously utilized glucose most likely has no value in ascribing a task to glycogen, since aglycaemia is an artificial experimental process, and hypoglycemia is an iatrogenic consequence of insulin therapy that didn't exist previous to 1922 (Frier et al., 2014), thus cannot be glycogen’s major healthy flow blood product position. However supplying lactate to axons when tissue energy demand increases implies a novel role for mind glycogen and suggests it acts as an vitality buffer quite than an energy reserve, whose position is to not single-handedly assist conduction, however to act in live performance with ambient ranges of normoglycemic glucose.
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